Pu Lab Publications 2022


2022 Original Manuscripts


  • Zhou, P., Zhang, Y., Sethi, I., Ye, L., Trembley, M. A., Cao, Y., Akerberg, B. N., Xiao, F., Zhang, X., Li, K., Jardin, B. D., Mazumdar, N., Ma, Q., He, A., Zhou, B. & Pu, W. T. GATA4 Regulates Developing Endocardium Through Interaction With ETS1. Circ. Res. (2022). doi:10.1161/CIRCRESAHA.120.318102. PDF. Supplement
  • Lu, F., Ma, Q., Xie, W., Liou, C. L., Zhang, D., Sweat, M. E., Jardin, B. D., Naya, F. J., Guo, Y., Cheng, H. & Pu, W. T. CMYA5 establishes cardiac dyad architecture and positioning. Nat. Commun. 13, 1–12 (2022). Link.
  • Zheng, Y., VanDusen, N. J., Butler, C. E., Ma, Q., King, J. S. & Pu, W. T. Efficient In Vivo Homology-Directed Repair Within Cardiomyocytes. Circulation 145, 787–789 (2022). PDF.
  • Guo, Y., Cao, Y., Jardin, B. D., Zhang, X., Zhou, P., Guatimosim, S., Lin, J., Chen, Z., Zhang, Y., Mazumdar, N., Lu, F., Ma, Q., Lu, Y.-W., Zhao, M., Wang, D.-Z., Dong, E. & Pu, W. T. Ryanodine receptor 2 (RYR2) dysfunction activates the unfolded protein response and perturbs cardiomyocyte maturation. Cardiovasc. Res. (2022). doi:10.1093/cvr/cvac077. Full Text.

2022 Reviews/Editorials

  • Thompson, R., Jefferies, J., Wang, S., Pu, W. T., Takemoto, C., Hornby, B., Heyman, A., Chin, M. T. & Vernon, H. J. Current and future treatment approaches for Barth syndrome. J. Inherit. Metab. Dis. 45, 17–28 (2022).
  • Pu, W. T. Experimental Models of Barth Syndrome. J. Inherit. Metab. Dis. (2021). doi:10.1002/jimd.12423. PDF.

2022 Collaborative Work

  • Yue, P., Zhang, Y., Liu, L., Zhou, K., Xia, S., Peng, M., Yan, H., Tang, X., Chen, Z., Zhang, D., Guo, J., Pu, W. T., Guo, Y., Hua, Y. & Li, Y. Yap1 modulates cardiomyocyte hypertrophy via impaired mitochondrial biogenesis in response to chronic mechanical stress overload. Theranostics 12, 7009–7031 (2022).
  • Sohn, J., Milosevic, J., Brouse, T., Aziz, N., Elkhoury, J., Wang, S., Hauschild, A., van Gastel, N., Cetinbas, M., Tufa, S. F., Keene, D. R., Sadreyev, R., Pu, W. T. & Sykes, D. B. A new murine model of Barth Syndrome neutropenia links TAFAZZIN deficiency to increased ER stress induced apoptosis. Blood Adv (2022). doi:10.1182/bloodadvances.2021005720
  • Yan, Y., Long, T., Su, Q., Wang, Y., Chen, K., Yang, T., Zhao, G., Ma, Q., Hu, X., Liu, C., Liao, X., Min, W., Li, S., Zhang, D., Yang, Y., Pu, W. T., Dong, Y., Wang, D.-Z., Chen, Y. & Huang, Z.-P. Cardiac ISL1-Interacting Protein, a Cardioprotective Factor, Inhibits the Transition From Cardiac Hypertrophy to Heart Failure. Front Cardiovasc Med 9, 857049 (2022).
  • Akerberg, A. A., Trembley, M., Butty, V., Schwertner, A., Zhao, L., Beerens, M., Liu, X., Mahamdeh, M., Yuan, S., Boyer, L., MacRae, C., Nguyen, C., Pu, W. T., Burns, C. E. & Burns, C. G. RBPMS2 Is a Myocardial-Enriched Splicing Regulator Required for Cardiac Function. Circ. Res. 131, 980–1000 (2022).